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Expression is lost in many cancer cells [44-46], and reintroduction of the protein often results in cell death [21,46,47]. Similarly, our results show that IGFBP3 expression is not detectable in GIST-T1 cells but overexpression leads to loss of cell viability. Indeed, the growth inhibitory and pro-apoptotic effects of IGFBP3 are well established in a variety of in vitro and in vivo cancer models.
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Ctional as long as either of the two MAPKKKs is catalytically active. Although less crucial, the MAPKKK heteromer also appears to contribute to prompt activation of the MAPK cascade upon oxidative stress (Supplemental Figure S2; Shiozaki et al., 1998).FIGURE 2: The Wis4 and Win1 MAPKKKs form a heteromer complex. (A) win1:myc and wis4:myc strains were used to monitor the expression levels of the MA
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Mined intents, 1987?011, percentage with 95 CI, by sexUndetermined 2 909 24.3 (22.8-25.9) 31.7 (30.0-33.4) 28.9 (27.3-30.6) 15.1 (13.8-16.4) 2 909 15.4 (14.1-16.7) 84.6 (83.3-85.9) 1 283 39.4 (36.8-42.1) 43.7 (41.0-46.4) 16.8 (14.8-18.9) 2 014 5.9 (4.9-6.9) 11 (9.6-12.4) 14.3 (12.8-15.8) 68.9 (66.9-70.9) 2 014 13.2 (11.7-14.7) 22.3 (20.5-24.1) 26.7 (24.8-28.6) 37.8 (35.7-39.9) 2 014 10.5 (9.2-11.
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Ctional as long as either of the two MAPKKKs is catalytically active. Although less crucial, the MAPKKK heteromer also appears to contribute to prompt activation of the MAPK cascade upon oxidative stress (Supplemental Figure S2; Shiozaki et al., 1998).FIGURE 2: The Wis4 and Win1 MAPKKKs form a heteromer complex. (A) win1:myc and wis4:myc strains were used to monitor the expression levels of the MA
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